transient global amnesia driving restriction

Is there any more recognition of the role of statins in transient global amnesia?Ī. The doctor said I will probably never have any recall of the day I missed, and I still don’t know what happened then. My doctors denied that atorvastatin could have had anything to do with this complete blank of my memory. I had a battery of tests, but all were negative. I had been on atorvastatin for only a few days when I was taken to the emergency room after a 12-hour episode of transient global amnesia. A Reader Describes Global Amnesia after Atorvastatin:

To the surprise of many health professionals and patients, statins can occasionally trigger something called transient global amnesia ( TGA).

A few medications can cause amnesia, including narcotics and benzodiazepines.

After brain trauma, some people cannot remember who they are or what happened recently. It is a kind of memory loss that can vary in both nature and intensity. Clinical use of 7 T will increase the number of patients having their TGA diagnosis supported by MRI, which can be especially useful in patients with negative 1.5/3 T MRI and low clinical certainty.ħ T 7 T MRI DWI lesion Hippocampal lesion TGA.No doubt you have heard of amnesia. Seven-Tesla MRI showed both a statistically significant increase in the total number of detected hippocampal DWI lesions and the proportion of patients with at least one hippocampal DWI lesion supporting the TGA diagnosis compared to 1.5/3 T. Both neuroradiologists found the hippocampal DWI lesions at 7 T to have higher conspicuity and be easier to categorize as true findings compared to 1.5/3 T. At 1.5/3 T, nine hippocampal DWI lesions were detected, which increased to 19 at 7 T, giving an increased detection rate of 111% (p = 0.002). At 1.5/3 T, six patients (46%) were found to have at least one hippocampal DWI-lesions supporting the TGA diagnosis, which increased to 11 patients (85%) when examined at 7 T (p = 0.03). The 7 T MRIs were performed a median of 15 h after the 1.5/3 T MRIs (range 1-28 h). Thirteen TGA patients with a median age of 68.5 years (range 46-77 years) were included and imaged at 1.5/3 T (median 17 h after onset of symptoms, range 3-23 h) and 7 T (median 23 h after onset, range 15-46 h). Patients with a clinical diagnosis consistent with TGA and a 1.5/3 T MRI underwent an additional 7 T MRI when the 7 T system was available for clinical use, thus serving as their own controls. To assess the ability of 7 T MRI to detect hippocampal DWI lesions in the acute phase of TGA compared to 1.5 T/3 T MRI.

7 Department of Circulation and Medical Imaging, Faculty of Medicine and Health Sciences, Norwegian University of Science and Technology, Trondheim, Norway.

6 Department of Physics, Faculty of Natural Sciences, Norwegian University of Science and Technology, Trondheim, Norway.

Olavs Hospital, Trondheim University Hospital, Trondheim, Norway.

5 Department of Radiology and Nuclear Medicine, St.

4 Department of Neuromedicine and Movement Science, Norwegian University of Science and Technology, Trondheim, Norway.

3 Department of Neurology and Clinical Neurophysiology, St. 2 Department of Radiology and Nuclear Medicine, St.

1 Department of Circulation and Medical Imaging, Faculty of Medicine and Health Sciences, Norwegian University of Science and Technology, Trondheim, Norway.

Transient global amnesia driving restriction